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Fournisseur:  Merck
Description:   Organic Standard, Diuron, Type de conditionnement: Flacon verre pour solides
UOM:  1 * 250 mg

Fournisseur:  Merck
Description:   Organic Standard, Ethephon, Type de conditionnement: Flacon verre pour solides
UOM:  1 * 250 mg

Fournisseur:  Merck
Description:   Organic Standard, Ethalfluralin, Type de conditionnement: Flacon verre pour solides
UOM:  1 * 250 mg

Fournisseur:  Merck
Description:   Organic Standard, Eugenol, Type de conditionnement: Flacon verre pour solides
UOM:  1 * 250 mg

Fournisseur:  Merck
Description:   Organic Standard, Dichlorvos, Type de conditionnement: Flacon verre pour solides
UOM:  1 * 250 mg

Fournisseur:  Merck
Description:   Organic Standard, Dinobuton, Type de conditionnement: Flacon verre pour solides
UOM:  1 * 250 mg

Fournisseur:  Merck
Description:   Organic Standard, β-HCH, Type de conditionnement: Flacon verre pour solides
UOM:  1 * 100 mg

Fournisseur:  Merck
Description:   Organic Standard, Triazoxid, Type de conditionnement: Flacon verre pour solides
UOM:  1 * 100 mg
Numéro de catalogue: (35588-100MG)

Fournisseur:  Merck
Description:   Organic Standard, PCB No 5, Type de conditionnement: Glass bottle for solids
UOM:  1 * 100 mg
Numéro de catalogue: (BOSSBS-8325R)

Fournisseur:  Bioss
Description:   Cell cycle progression is subject to arrest at G1 and G2 checkpoints in response to DNA damage, presumably to allow time for DNA repair prior to entry into S and M phase, respectively. The p53 tumor suppressor is required for one such G1 checkpoint and functions to upregulate expression of GADD 45 and the mitotic inhibitory protein p21. GADD 45 stimulates DNA excision repair in vitro and inhibits entry of cells into S phase, and it apparently acts in concert with GADD 153 in inducing growth arrest. A related DNA-damage inducible gene, GADD 34 synergizes with GADD 45 or GADD 153 in suppressing cell growth. PEG-3 (progression elevated gene-3) shares significant homology with GADD 34 and is inducible by DNA damage. An additional GADD related gene, PA26, is a possible target of p53. Three isoforms of PA26 have been identified as PA26-T1, PA26-T2 and PA26-T3.
UOM:  1 * 100 µl
Numéro de catalogue: (BOSSBS-8325R-CY3)

Fournisseur:  Bioss
Description:   Cell cycle progression is subject to arrest at G1 and G2 checkpoints in response to DNA damage, presumably to allow time for DNA repair prior to entry into S and M phase, respectively. The p53 tumor suppressor is required for one such G1 checkpoint and functions to upregulate expression of GADD 45 and the mitotic inhibitory protein p21. GADD 45 stimulates DNA excision repair in vitro and inhibits entry of cells into S phase, and it apparently acts in concert with GADD 153 in inducing growth arrest. A related DNA-damage inducible gene, GADD 34 synergizes with GADD 45 or GADD 153 in suppressing cell growth. PEG-3 (progression elevated gene-3) shares significant homology with GADD 34 and is inducible by DNA damage. An additional GADD related gene, PA26, is a possible target of p53. Three isoforms of PA26 have been identified as PA26-T1, PA26-T2 and PA26-T3.
UOM:  1 * 100 µl
Numéro de catalogue: (BOSSBS-3091R-CY5)

Fournisseur:  Bioss
Description:   The cell division control protein cdc2, also known as cyclin dependent kinase 1 (Cdk1) or p34/cdk1, plays a key role in the control of the eukaryotic cell cycle, where it is required for entry into S phase and mitosis. Cdc2 exists as a complex with both cyclin A and cyclin B. The best characterized of these associations is the Cdc2 p34 cyclin B complex, which is required for the G2 to M phase transition. Activation of Cdc2 is controlled at several steps including cyclin binding and phosphorylation of threonine 161. However, the critical regulatory step in activating cdc2 during progression into mitosis appears to be dephosphorylation of Tyr15 and Tyr14. Phosphorylation at Tyr15 and inhibition of Cdc2 is carried out by WEE1 and MIK protein kinases while Tyr15 dephosphorylation and activation of Cdc2 is carried out by the cdc25 phosphatase. The isoform CDC2deltaT is found in breast cancer tissues. Furthermore, cdc2/Cdk1 is a key mediator of neuronal cell death in brain development and degeneration.
UOM:  1 * 100 µl
Numéro de catalogue: (BOSSBS-8325R-CY7)

Fournisseur:  Bioss
Description:   Cell cycle progression is subject to arrest at G1 and G2 checkpoints in response to DNA damage, presumably to allow time for DNA repair prior to entry into S and M phase, respectively. The p53 tumor suppressor is required for one such G1 checkpoint and functions to upregulate expression of GADD 45 and the mitotic inhibitory protein p21. GADD 45 stimulates DNA excision repair in vitro and inhibits entry of cells into S phase, and it apparently acts in concert with GADD 153 in inducing growth arrest. A related DNA-damage inducible gene, GADD 34 synergizes with GADD 45 or GADD 153 in suppressing cell growth. PEG-3 (progression elevated gene-3) shares significant homology with GADD 34 and is inducible by DNA damage. An additional GADD related gene, PA26, is a possible target of p53. Three isoforms of PA26 have been identified as PA26-T1, PA26-T2 and PA26-T3.
UOM:  1 * 100 µl

Fournisseur:  Merck
Description:   Organic Standard, Propargite, Type de conditionnement: Flacon verre pour solides
UOM:  1 * 100 mg

Fournisseur:  Merck
Description:   Organic Standard, Chlorfenvinphos, Type de conditionnement: Flacon verre pour solides
UOM:  1 * 250 mg

Fournisseur:  Bioss
Description:   The protein encoded by this gene is a component of the multi-subunit protein complex EIF4F. This complex facilitates the recruitment of mRNA to the ribosome, which is a rate-limiting step during the initiation phase of protein synthesis. The recognition of the mRNA cap and the ATP-dependent unwinding of 5'-terminal secondary structure is catalyzed by factors in this complex. The subunit encoded by this gene is a large scaffolding protein that contains binding sites for other members of the EIF4F complex. A domain at its N-terminus can also interact with the poly(A)-binding protein, which may mediate the circularization of mRNA during translation. Alternative splicing results in multiple transcript variants, some of which are derived from alternative promoter usage. [provided by RefSeq].
UOM:  1 * 100 µl
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Le stock de cet article est limité mais peut être disponible dans un entrepôt proche de vous. Merci de vous assurer que vous êtes connecté sur le site afin que le stock disponible soit affiché. Si l'call est toujours affiché et vous avez besoin d'aide, s'il vous plaît appelez-nous au 016 385 011
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