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acids+and+salts+high+purity
Numéro de catalogue:
(EHERC16813610)
Fournisseur:
EHRENSTORFER GMBH
Description:
Riboflavine-5'-phosphate sodium salt
UOM:
1 * 0,25 g
Numéro de catalogue:
(AATB21144)
Fournisseur:
AAT BIOQUEST INC
Description:
Cal-520® provides a robust homogeneous fluorescence-based assay tool for detecting intracellular calcium mobilization.
UOM:
1 * 500 µG
New Product
Numéro de catalogue:
(BOSSBS-13627R-CY7)
Fournisseur:
Bioss
Description:
The family of voltage-dependent chloride channels (CLCs) regulate cellular trafficking of chloride ions, a critical component of all living cells. CLCs regulate excitability in muscle and nerve cells, aid in organic solute transport, and maintain cellular volume. CLC-KA is a kidney-specific chloride channel that mediates transepithelial chloride transport in the thin ascending limb of the Henle loop in the inner medulla. CLC-KA plays a crucial role in urine concentration. The gene encoding human CLC-KA maps to chromosome 1p36. Mutations in this gene may be associated with nephrogenic diabetes insipidus in those cases where mutations in the vasopressin V2 receptor and the AQP2 water channel are lacking. CLC-KB mediates basolateral chloride ion efflux in the thick ascending limb and in more distal nephron segments. The gene encoding human CLC-KB maps to chromosome 1p36. Mutations in this gene cause type III Barter’s syndrome which is characterized by renal salt-wasting and low blood pressure.
UOM:
1 * 100 µl
Fournisseur:
Merck Millipore (Calbiochem)
Description:
Dextran sulphate sodium salt, Calbiochem®
Numéro de catalogue:
(BOSSBS-13627R-FITC)
Fournisseur:
Bioss
Description:
The family of voltage-dependent chloride channels (CLCs) regulate cellular trafficking of chloride ions, a critical component of all living cells. CLCs regulate excitability in muscle and nerve cells, aid in organic solute transport, and maintain cellular volume. CLC-KA is a kidney-specific chloride channel that mediates transepithelial chloride transport in the thin ascending limb of the Henle loop in the inner medulla. CLC-KA plays a crucial role in urine concentration. The gene encoding human CLC-KA maps to chromosome 1p36. Mutations in this gene may be associated with nephrogenic diabetes insipidus in those cases where mutations in the vasopressin V2 receptor and the AQP2 water channel are lacking. CLC-KB mediates basolateral chloride ion efflux in the thick ascending limb and in more distal nephron segments. The gene encoding human CLC-KB maps to chromosome 1p36. Mutations in this gene cause type III Barter’s syndrome which is characterized by renal salt-wasting and low blood pressure.
UOM:
1 * 100 µl
Numéro de catalogue:
(J64329.03)
Fournisseur:
Thermo Scientific
Description:
Uridine-5'-diphosphate disodium salt 98%
UOM:
1 * 1 g
Numéro de catalogue:
(BOSSBS-10307R-CY3)
Fournisseur:
Bioss
Description:
The family of voltage-dependent chloride channels (CLCs) regulate cellular trafficking of chloride ions, a critical component of all living cells. CLCs regulate excitability in muscle and nerve cells, aid in organic solute transport and maintain cellular volume. The genes encoding human CLC-1 through CLC-7 map to chromosomes 7q32, 3q28, 4q32, Xp22.3, Xp11.23-p11.22, 1p36 and 16p13, respectively. CLC1 is highly expressed in skeletal muscle. Mutations in the gene encoding CLC1 lead to myotonia, an inheritable disorder characterized by muscle stiffness and renal salt wasting. CLC2 is highly expressed in the epithelia of several organs including lung, which suggests CLC2 may be a possible therapeutic target for cystic fibrosis. CLC3 expression is particularly abundant in neuronal tissue, while CLC4 expression is evident in skeletal and cardiac muscle as well as brain. Mutations in the gene encoding CLC5 lead to Dent’s disease, a renal disorder characterized by proteinuria and hypercalciuria. CLC6 and CLC7 are broadly expressed in several tissues including testis, kidney, brain and muscle.
UOM:
1 * 100 µl
Numéro de catalogue:
(BOSSBS-10307R-CY5)
Fournisseur:
Bioss
Description:
The family of voltage-dependent chloride channels (CLCs) regulate cellular trafficking of chloride ions, a critical component of all living cells. CLCs regulate excitability in muscle and nerve cells, aid in organic solute transport and maintain cellular volume. The genes encoding human CLC-1 through CLC-7 map to chromosomes 7q32, 3q28, 4q32, Xp22.3, Xp11.23-p11.22, 1p36 and 16p13, respectively. CLC1 is highly expressed in skeletal muscle. Mutations in the gene encoding CLC1 lead to myotonia, an inheritable disorder characterized by muscle stiffness and renal salt wasting. CLC2 is highly expressed in the epithelia of several organs including lung, which suggests CLC2 may be a possible therapeutic target for cystic fibrosis. CLC3 expression is particularly abundant in neuronal tissue, while CLC4 expression is evident in skeletal and cardiac muscle as well as brain. Mutations in the gene encoding CLC5 lead to Dent’s disease, a renal disorder characterized by proteinuria and hypercalciuria. CLC6 and CLC7 are broadly expressed in several tissues including testis, kidney, brain and muscle.
UOM:
1 * 100 µl
Fournisseur:
Thermo Scientific
Description:
3,3',5'-Triiodo-L-thyronine sodium salt
Numéro de catalogue:
(BOSSBS-13627R-CY3)
Fournisseur:
Bioss
Description:
The family of voltage-dependent chloride channels (CLCs) regulate cellular trafficking of chloride ions, a critical component of all living cells. CLCs regulate excitability in muscle and nerve cells, aid in organic solute transport, and maintain cellular volume. CLC-KA is a kidney-specific chloride channel that mediates transepithelial chloride transport in the thin ascending limb of the Henle loop in the inner medulla. CLC-KA plays a crucial role in urine concentration. The gene encoding human CLC-KA maps to chromosome 1p36. Mutations in this gene may be associated with nephrogenic diabetes insipidus in those cases where mutations in the vasopressin V2 receptor and the AQP2 water channel are lacking. CLC-KB mediates basolateral chloride ion efflux in the thick ascending limb and in more distal nephron segments. The gene encoding human CLC-KB maps to chromosome 1p36. Mutations in this gene cause type III Barter’s syndrome which is characterized by renal salt-wasting and low blood pressure.
UOM:
1 * 100 µl
Fournisseur:
AAT BIOQUEST INC
Description:
Calcium measurement is critical for numerous biological investigations.
Numéro de catalogue:
(AATB20404)
Fournisseur:
AAT BIOQUEST INC
Description:
A vast majority of the existing magnesium ion indicators are based on tricarboxylate APTRA chelator derived from the popular tetracarboxylate BAPTA calcium ion chelator.
UOM:
1 * 500 µG
New Product
Fournisseur:
AAT BIOQUEST INC
Description:
Cal-520® provides a robust homogeneous fluorescence-based assay tool for detecting intracellular calcium mobilization.
Numéro de catalogue:
(TCIAD4228-25MG)
Fournisseur:
TCI
Description:
Dibutyryl-cAMP sodium salt ≥97.0% (par HPLC)
UOM:
1 * 25 mg
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est toujours affiché et vous avez besoin d'aide, s'il vous plaît appelez-nous au 016 385 011
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